Order ID | 53563633773 |
Type | Essay |
Writer Level | Masters |
Style | APA |
Sources/References | 4 |
Perfect Number of Pages to Order | 5-10 Pages |
Trigeminal Neuralgia Case Study Discussion Paper
Introduction
[Your introduction is an explanation as to what the paper is going to be about; in this case it is the function of the Trigeminal Nerve, Trigeminal Neuralgia and the treatment of Trigeminal Neuralgia. Please dedicate at least one page, describing the function of the Trigeminal Nerve.
When you start a new thought and want to start a new paragraph, the APA format will automatically indent the first sentence of your new paragraph when you tap the “enter” button on your keyboard.
When you discuss a term or title that may be paraphrased through the use of an acronym or shortened name, make sure to write out the full version of the term or title and put the shorted version or acronym right behind it, (in brackets).]
Trigeminal Neuralgia
[Please dedicate at least one page to the discussion of what Trigeminal Neuralgia is; what causes the disorder.]
Treatment
[Please dedicate at least one page to the discussion of how Trigeminal Neuralgia is treated (i.e., surgery, medications, therapy, etc.) and why it is important for dental hygienist to know and recognize it or question their patients about it.]
[Remember, any information that you could not possibly know, unless you wrote the study yourself, such as numerical data, dates, etc., must be referenced in APA Style ®. (Last Name, Year)]
References
Last Name, F. M. (Year). Article Title. Journal Title, Pages From – To.
Last Name, F. M. (Year). Book Title. City Name: Publisher Name.
Hairy Tongue Oral Pathology Presentation
Oral Pathology Case presentation Criteria
Oral pathology involves the study of oral lesions and diseases. Upon completion of this oral pathology case study, the student should be well versed and be knowledgeable of the specific topic presented. You may choose either a written case presentation, or a digital presentation. Discuss a topic from one of the chapters in the textbook. Examples could be a lesion, disease, genetic condition, etc. You will need to include: a description of the lesion or disease, a discussion of the diagnostic process for that lesion/disease, and the treatment and prognosis.
PHA Biology Discussion Questions
I’m working on a biology multi-part question and need an explanation and answer to help me learn.
With respect to thermodynamics, why can biosynthetic pathways not be simply considered the reverse of corresponding catabolic pathways?
Compare the net equations of glycolysis and gluconeogenesis and explain why the synthesis of glucose is so costly.
Why is the biosynthesis of glucose so important in mammals?
List the following:
Most common precursors for glucose biosynthesis
Organs which use glucose as their primary energy source
Organs or tissues where gluconeogenesis takes place
Explain in detail how both glycolysis and gluconeogenesis can be irreversible cellular processes.
What drives the conversion of fructose 1, 6- bisphosphate to fructose 6 -phosphate?
Describe the role of malate transport in gluconeogenesis.
How does fatty acid oxidation contribute to gluconeogenesis? Under what conditions do amino acids get converted to glucose?
What are the effects of G6P, F1,6-BP, F2,6-BP on glycolysis and gluconeogenesis?
What are the effects of cAMP, AMP, ATP, citrate and acetyl-CoA on glycolysis and gluconeogenesis?
How many ATP equivalents are required to synthesize a glucose from two pyruvate in order to bypass the irreversible steps in glycolysis?
Why are sugar nucleotides suitable for biosynthetic reactions?
How does F2,6-BP control the conversion of F6P to F1,6-BP or the reverse?
In the final stages of gluconeogenesis, glucose-6-phosphatase converts glucose-6-phosphate to free glucose and inorganic phosphate. Where does this occur?
Read the article below. This study addresses the synthesis of a trisaccharide that humans do not metabolize. Why are studies such as this and others which examine bacterial systems for enzymatic activity on carbohydrates ultimately beneficial to addressing human health concerns?
Perrin V, Fenet B, Prally J, Lecroix F, and Ta CD. (2000) Carbohydrate Research, 325 (3): 202-210.
Hepatocytes are an important model for in vitro study of metabolism. Read the article below and describe how the supplementation of cell culture media with amino acids and hormones affected the activity of hepatocytes and the occurrence of futile cycles. Chan C, Berthiaume F, Lee K, and Yarmush M. (2003) Metabolic Engineering, 5 (1): 1-15.
Solubilization of dietary lipids, achieved by mixing bile salts and degrading them with pancreatic lipases, is required for transport across the intestine where the free fatty acid and the lysophospholipid can then be resynthesized into triacylglycerols in the intestinal mucosal cells. Why is this necessary?
Describe how and why the above differs from the transport of fatty acids into the mitochondria?
Fatty acid oxidation occurs in the mitochondrial matrix. In review, where did the oxidative catabolism of glucose take place?
How are triacylglycerols functionally sequestered within adipose cells prior to a hormonal signal which causes them to be released into the bloodstream?
Explain in detail how different types of lipids are transported in blood to AND from adipose tissue.
What is the energetic cost of fatty acyl-CoA formation and where does this fatty acid activation take place?
Explain the roles of the two pools of coenzyme A and the processes utilized to keep these pools separate.
What types of reactions make up the 4 basic steps of β-oxidation?
Describe the enzyme complexes that receive electrons form the reduced electron carriers.
How many molecules of ATP are generated by the reoxidation of each type of electron carrier?
Compare saturated and unsaturated fatty acid oxidation with respect to the amount of ATP formed.
Explain how odd-chain fatty acid oxidation differs from even chain fatty acid oxidation beginning with the 5 carbon fatty acyl-CoA encountered in the last pass through the β-oxidation sequence?
Where in the cell are Ketone bodies made, from which metabolite are they made, and how and where are ketone bodies used?
Outline the oxidation steps that occur in omega ( ω ) oxidation.
We have touched on saturated/unsaturated and even-/odd-numbered chain fatty acids, but dietary consumption also includes branched chain fatty acids. Locate and read the following article, and answer the questions below: Mukherji M, Schofield CJ, Wierzbicki AS, Jansen GA, Wanders RJA, and Lloyd M. (2003). Progress in Lipid Research, 42 (5): 359-376.
In which organelle not previously mentioned in this assignment does metabolism of some branched-chain fatty acids occur?
According to your text and this paper, what is the primary pathway for oxidation of long chain fatty acids in this
organelle?
What name is given the oxidative pathway for branched-chain fatty acids?
What, in general terms, is an oxygenase?
In the proposed mechanism for 2-hydroxyphytanoyl-CoA lyase, what two products result from the attack of TPP on thioester?
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